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Cholecystokinin-like peptide (DSK) in Drosophila, not only for satiety signaling
Stockholms universitet, Naturvetenskapliga fakulteten, Zoologiska institutionen.
Rekke forfattare: 22014 (engelsk)Inngår i: Frontiers in Endocrinology, ISSN 1664-2392, E-ISSN 1664-2392, Vol. 5, artikkel-id 219Artikkel, forskningsoversikt (Fagfellevurdert) Published
Abstract [en]

Cholecystokinin (CCK) signaling appears well conserved over evolution. In Drosophila, the CCK-like sulfakinins (DSKs) regulate aspects of gut function, satiety and food ingestion, hyperactivity and aggression, as well as escape-related locomotion and synaptic plasticity during neuromuscular junction development. Activity in the DSK-producing neurons is regulated by octopamine. We discuss mechanisms behind CCK function in satiety, aggression, and locomotion in some detail and highlight similarities to mammalian CCK signaling.

sted, utgiver, år, opplag, sider
2014. Vol. 5, artikkel-id 219
Emneord [en]
neuropeptide, peptide hormone, aggression, feeding, intestinal function, locomotion
HSV kategori
Identifikatorer
URN: urn:nbn:se:su:diva-161688DOI: 10.3389/fendo.2014.00219ISI: 000209749800217PubMedID: 25566191OAI: oai:DiVA.org:su-161688DiVA, id: diva2:1260923
Tilgjengelig fra: 2018-11-05 Laget: 2018-11-05 Sist oppdatert: 2018-11-05bibliografisk kontrollert

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