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Cholecystokinin-like peptide (DSK) in Drosophila, not only for satiety signaling
Stockholm University, Faculty of Science, Department of Zoology.ORCID iD: 0000-0002-1147-7766
Number of Authors: 22014 (English)In: Frontiers in Endocrinology, ISSN 1664-2392, E-ISSN 1664-2392, Vol. 5, article id 219Article, review/survey (Refereed) Published
Abstract [en]

Cholecystokinin (CCK) signaling appears well conserved over evolution. In Drosophila, the CCK-like sulfakinins (DSKs) regulate aspects of gut function, satiety and food ingestion, hyperactivity and aggression, as well as escape-related locomotion and synaptic plasticity during neuromuscular junction development. Activity in the DSK-producing neurons is regulated by octopamine. We discuss mechanisms behind CCK function in satiety, aggression, and locomotion in some detail and highlight similarities to mammalian CCK signaling.

Place, publisher, year, edition, pages
2014. Vol. 5, article id 219
Keywords [en]
neuropeptide, peptide hormone, aggression, feeding, intestinal function, locomotion
National Category
Endocrinology and Diabetes
Identifiers
URN: urn:nbn:se:su:diva-161688DOI: 10.3389/fendo.2014.00219ISI: 000209749800217PubMedID: 25566191OAI: oai:DiVA.org:su-161688DiVA, id: diva2:1260923
Available from: 2018-11-05 Created: 2018-11-05 Last updated: 2019-12-10Bibliographically approved

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