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Thermogenesis challenges the adipostat hypothesis for body-weight control.
Stockholms universitet, Naturvetenskapliga fakulteten, Wenner-Grens institut, Avdelningen för fysiologi.
Stockholms universitet, Naturvetenskapliga fakulteten, Wenner-Grens institut, Avdelningen för fysiologi.ORCID-id: 0000-0003-2070-1587
2009 (engelsk)Inngår i: Proceedings of the Nutrition Society, ISSN 0029-6651, E-ISSN 1475-2719, Vol. 68, nr 4, s. 401-7Artikkel i tidsskrift (Fagfellevurdert) Published
Abstract [en]

According to the adipostat hypothesis for body-weight control, alterations in body weight should always be compensated by adequate alterations in food intake and thermogenesis. Thus, increased thermogenesis should not be able to counteract obesity because food intake would be increased. However evidence is presented here that thermogenesis in different forms (through artificial uncouplers, exercise, cold exposure) may counteract obesity and is not always fully compensated by increased food intake. Correspondingly, a decreased capacity for metaboloregulatory thermogenesis (i.e. non-functional brown adipose tissue) may in itself lead to obesity. This is evident in mice and may be valid for human subjects, as a substantial proportion of adults possess brown adipose tissue, and those with less or without brown adipose tissue would seem to be more prone to obesity. Thus, increased thermogenesis may counteract obesity, without dietary intervention.

sted, utgiver, år, opplag, sider
2009. Vol. 68, nr 4, s. 401-7
HSV kategori
Forskningsprogram
fysiologi; fysiologi
Identifikatorer
URN: urn:nbn:se:su:diva-32934DOI: 10.1017/S0029665109990255ISI: 000271732800008PubMedID: 19775494OAI: oai:DiVA.org:su-32934DiVA, id: diva2:281994
Tilgjengelig fra: 2009-12-18 Laget: 2009-12-18 Sist oppdatert: 2022-02-25bibliografisk kontrollert

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