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Covert coping with unfair treatment at work and risk of incident myocardial infarction and cardiac death among men: prospective cohort study
Stockholms universitet, Samhällsvetenskapliga fakulteten, Stressforskningsinstitutet.ORCID-id: 0000-0001-8433-2405
Stockholms universitet, Samhällsvetenskapliga fakulteten, Stressforskningsinstitutet.ORCID-id: 0000-0002-8806-5698
Stockholms universitet, Samhällsvetenskapliga fakulteten, Stressforskningsinstitutet.ORCID-id: 0000-0002-3845-3545
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2011 (engelsk)Inngår i: Journal of Epidemiology and Community Health, ISSN 0143-005X, E-ISSN 1470-2738, Vol. 65, nr 5, s. 420-425Artikkel i tidsskrift (Fagfellevurdert) Published
Abstract [en]

Background: Covert coping with unfair treatment at work-occurring when an employee does not show the "aggressor" that he/she feels unfairly treated-has been found to be associated with cardiovascular risk factors. This study examined whether covert coping also predicts incident coronary heart disease.

Methods: A prospective cohort study (the WOLF Stockholm study) of workplaces in the Stockholm area, Sweden. The participants were 2755 men with no history of myocardial infarction at baseline screening in 1992-1995. The main outcome measure was hospitalisation due to myocardial infarction or death from ischaemic heart disease until 2003 obtained from national registers (mean follow-up 9.8±0.9 years).

Results: Forty-seven participants had myocardial infarction or died from ischaemic heart disease during follow-up. After adjustment for age, socioeconomic factors, risk behaviours, job strain and biological risk factors at baseline, there was a dose-response relationship between covert coping and risk of incident myocardial infarction or cardiac death (p for trend=0.10). Men who frequently used covert coping had a 2.29 (95% CI 1.00 to 5.29) times higher risk than those who did not use coping. Restricting the analysis to direct coping behaviours only strengthened this association (p for trend=0.02).

Conclusions: In this study, covert coping is strongly related to increased risk of hard-endpoint cardiovascular disease.

sted, utgiver, år, opplag, sider
2011. Vol. 65, nr 5, s. 420-425
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Identifikatorer
URN: urn:nbn:se:su:diva-56564DOI: 10.1136/jech.2009.088880ISI: 000289079700009PubMedID: 19934168Lokal ID: P2857OAI: oai:DiVA.org:su-56564DiVA, id: diva2:411723
Tilgjengelig fra: 2011-04-19 Laget: 2011-04-19 Sist oppdatert: 2022-03-16bibliografisk kontrollert

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